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Glaucoma: Open-Angle, Closed-Angle, Symptoms, Pharmacology, Pathophysiology Nursing NCLEX

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0:00hey everyone it's sarah with

0:01registerednessrn.com and in this video

0:03we're going to talk about glaucoma

0:05and as always whenever you get done

0:06watching this youtube lecture you can

0:08access the free quiz that will test you

0:10on this condition

0:12so let's get started glaucoma is a set

0:14of eye diseases that is caused by

0:16increased intraocular pressure and

0:19whenever this happens it results in

0:21optic nerve damage so what does our

0:24optic nerve do

0:25well this nerve helps transmit

0:27information that we see with our eyes

0:30to our brains so it plays a huge role in

0:32giving us our vision

0:34so if this nerve becomes damaged the

0:36person is going to have vision loss

0:38that can eventually lead to permanent

0:40blindness for instance if this patient

0:42had

0:43ankle glaucoma over time eventually they

0:46will notice that they're losing their

0:47peripheral vision which is going to give

0:49them like tunnel vision

0:50and if they don't get this treated where

0:52they're getting their interocular

0:54pressure

0:54lowered it can eventually lead to

0:56permanent blindness

0:58so in this lecture we're going to talk

0:59about two types of glaucoma

1:02the first type we're going to talk about

1:03is called open angle glaucoma

1:05and this type is the most common form of

1:08glaucoma

1:09now the thing you want to remember about

1:10this type is that its signs and symptoms

1:12are very subtle

1:14the patient's not going to really know

1:15that they have this type of glaucoma

1:17until the disease has really advanced

1:19so we refer to this type as the silent

1:22type

1:23the next type we're going to talk about

1:24is called angle closure glaucoma now you

1:27may also hear this referred to as

1:29narrow angle glaucoma or closed angle

1:32glaucoma

1:33all those terms refer to this type of

1:34glaucoma now this type of glaucoma is

1:37very

1:37rare and its signs and symptoms the

1:39patient is definitely going to notice

1:42them and whenever the patient has this

1:44type of glaucoma

1:45it requires immediate emergency

1:48treatment so as a nurse it's very

1:49essential that you know the differences

1:51between the signs and symptoms

1:53with open angle and ankle closure

1:55glaucoma

1:56now with all types of glaucoma you

1:58really want to stress to the patient

2:00that early detection

2:01is key because this disease can be

2:05detected on an eye exam

2:06that checks the intraocular pressure a

2:09little bit later we're going to talk

2:10about that specific exam

2:12so first let's talk about the

2:13pathophysiology of glaucoma

2:15and to help us truly understand this

2:18disease process

2:18we have to identify the key players that

2:21play a role in the pathophysiology

2:23so our key players are the intraocular

2:25pressure the iop

2:27and the aqueous humor so first let's

2:30talk about iop

2:31what is this well to help us understand

2:33that let's take that word apart

2:35intra means within and ocular means i so

2:38when we put that together

2:39we get the fluid pressure within the eye

2:42and the fluid that we're specifically

2:43talking about is the aqueous

2:45humor so intraocular pressure is

2:48calculated by the production rate and

2:51the drainage rate of this aqueous humor

2:54now aqueous humor is produced in the eye

2:56specifically by the ciliary

2:58body and it drains out through the eye

3:00and it's going to go through the

3:01trabecular

3:02meshwork into like the drainage canals

3:04so if your production rate and your

3:06drainage rate are not equal

3:08you're going to get increased

3:09interocular pressure but how in the

3:11world do we know a patient's iop

3:13well it can be detected through an eye

3:15exam and they can use a device called a

3:17tonometer

3:18and this device will tell us what the

3:20patient's iop is

3:22now generally a normal iop in most

3:25people is about 10

3:29to 21 millimeters of

3:32mercury so now let's talk about aqueous

3:35humor

3:35so we have established that this fluid

3:37helps maintain our iop

3:39but let's talk about how it really flows

3:42through this

3:43eye and really compare that to open and

3:46angle closure

3:47glaucoma so here we have our eye and the

3:50view of this eye is if you were looking

3:52over someone and you took their eyeball

3:54and you just cut it in half

3:56and you were just looking at it so this

3:57is the view of the eye that we're

3:58looking at

3:59and here in the drawing we have in red

4:01this is the

4:02ciliary body and that produces again the

4:04aqueous humor

4:06we have the lens we have in purple this

4:09represents the

4:10flow path of the aqueous humor and then

4:13here in black

4:14we have the pupil opening and then the

4:17colored area in green

4:19is the iris which is the color part of

4:20the eye so our person has green eyes

4:24and then we have the cornea which is

4:26this area here in black

4:28and then we have the trabecular meshwork

4:31and schlemm's canal and that's how the

4:33aqueous humor is going to

4:34leave this part of the eye so let's talk

4:36about how it flows

4:38normally so the ciliary body produces

4:41the aqueous

4:42humor and it's going to go through the

4:45posterior chamber of the eye

4:47and this is found between the iris and

4:50the lens

4:51then this aqueous humor is going to go

4:53through the pupil opening

4:55and it's going to enter into the

4:56anterior chamber of the eye

4:59which is found with the cornea and

5:02the iris then the aqueous humerus should

5:05normally go down

5:06and we're hitting the drainage angles of

5:08the eye and i want you to remember

5:09drainage angle because that is really

5:11the key concept

5:12in understanding closed angle or angle

5:14closure glaucoma

5:16versus open angle glaucoma so it's going

5:18to go down through these

5:19drainage angles and it's going to hit

5:21the trabecular

5:22meshwork okay it's mesh work so this

5:26trabecular mesh work is like a strainer

5:28area

5:28so the fluid is going to go down through

5:30there and then it's going to go into

5:32schwim's canal

5:33and then it's going to hit the

5:34episcleral veins and that's how it

5:36normally

5:37should drain out of the eye but to help

5:39give you a better

5:40illustration let's look at this

5:42animation so here is a

5:44side view of the eye if you took it and

5:46you cut it in half

5:48encircled in red is the ciliary body

5:50which produces that aqueous humor it's

5:52going to flow out through that posterior

5:54chamber

5:54through the pupillary open through the

5:57anterior

5:58chamber and then it's going to go down

6:00into the trabecular meshwork into the

6:02schlemm's canal in the episclerial veins

6:04so as you can see this aqueous humor is

6:07easily just flowing through the

6:09eye but now let's talk about when this

6:11aqueous humor doesn't flow too great

6:13through the eye

6:14and it leads to increased intraocular

6:16pressure and we get damage to our optic

6:18nerve

6:19so first let's talk about open angle

6:21glaucoma so i pointed out these drainage

6:23angles

6:24and the drainage angle of the eye is

6:26formed

6:27with the cornea and the iris so hence

6:30it's a drainage angle it forms an angle

6:32between these two structures

6:34now in open angle glaucoma this angle

6:38is open so the aqueous humor

6:42can flow down to where it needs to go

6:44into the trabecular meshwork

6:46but the problem with open angle is not

6:49the drainage angle

6:50it's with the trabecular meshwork now i

6:53pointed out earlier

6:54to think of the mesh work as like a

6:56strainer because what happens is as we

6:59age

7:00this mesh work becomes less flexible

7:03it's less permeable it becomes thicker

7:05so it will become

7:07clogged so this aqueous humor cannot get

7:10through the trabecular meshwork

7:12and we get increased pressure and as you

7:14can see here

7:15when the trabecular mesh work is not

7:17working the fluid will start to collect

7:20in the eye and then you're going to get

7:22this increased pressure

7:24which is going to put pressure on that

7:26optic nerve and can cause

7:28vision damage so now let's talk about

7:30the signs and symptoms

7:31of open angle glaucoma well as i pointed

7:34out earlier the signs and symptoms are

7:36going to

7:36appear very subtly for that patient and

7:39whenever they actually notice

7:41like that tunnel vision where they're

7:42having that loss of that peripheral

7:44vision

7:44that means this disease has advanced so

7:47through early detection we can see some

7:49of these other signs and symptoms which

7:51lets us know that hey this patient may

7:52have open ankle glaucoma

7:54so really it's silent in its

7:56presentation and you may even

7:58hear it referred to as the thief of

8:00sight

8:01but typically these patients are not

8:02going to have pain with this type of

8:04glaucoma compared to

8:05angle closure glaucoma they're of course

8:08going to have increased

8:09intraocular pressure most patients will

8:12and

8:12this will be found through various

8:14readings now again how did we determine

8:16a patient's iop

8:18it was through a tonometer and whenever

8:22they

8:22look at the patient's drainage angles

8:24because you can look at the drainage

8:25angles through

8:26one of those eye exams they can use a

8:28gonioscope to assess that so that can

8:31tell us

8:31if they have angle closure or open angle

8:34and they may have what's called

8:36optic disc cupping and this can be

8:39assessed with an ophthalmoscope

8:41and you can look at the fundus of the

8:43eye

8:44and you can look at the optic disc to

8:46cup ratio

8:48and the optic disc is found on the

8:50fundus and in the middle of the disc

8:52is the optic cup so with chronic open

8:55ankle glaucoma the cup will become

8:57bigger compared to the disc and here

9:00on the right you will see a normal optic

9:03nerve

9:04notice the cup it's the bright part in

9:07the middle is smaller compared to the

9:09disc

9:09but here on the left the optic cup is a

9:12lot bigger compared to the disc now

9:14let's talk about angle closure glaucoma

9:16so again with these drainage angles we

9:18have the cornea and the iris and we have

9:21that angle

9:22and what's happened is that this angle

9:24has narrowed

9:25his hints close so this aqueous humor

9:28cannot get down into the trabecular

9:30meshwork into schlemm's canal and do its

9:32thing

9:33so we don't have a problem with

9:35trabecular's mesh work

9:36it's that this angle has closed off and

9:39what's going to happen is this aqueous

9:40humor is going to build up in

9:42on the eye and it's going to really

9:44severely

9:45hinder this optic nerve now when this

9:47type of glaucoma happens

9:49it is an emergency it's rare to happen

9:51but when it does it the person needs

9:53treatment immediately we're going to

9:54talk about those treatments a little bit

9:55later so what could cause this to happen

10:00well if a person already naturally has

10:03narrowed angles an anatomical defect

10:05that can cause it

10:06but medications can cause this that

10:08dilate

10:09that pupil because what it can do is it

10:11can push the lens or the iris forward

10:14and when you push that forward you close

10:16that angle

10:17and medication that can do that is like

10:20anticholinergics

10:21and you want to educate patients who are

10:23at risk for this to avoid those type of

10:24medications

10:25because you can get these like over the

10:27counter with like allergy medicines

10:28antihistamines

10:30or copd drugs or antidepressants so

10:34big risk with those now what are the

10:36signs and symptoms of this type of

10:38glaucoma

10:39well the patient is going to be able to

10:41notice them and they're not silent like

10:43with the other type of glaucoma

10:45so they can report severe eye pain

10:47they're going to have a lot of pain with

10:48this type of glaucoma

10:49they can also have nausea and vomiting

10:52they're going to have vision changes

10:53where their vision will be blurred from

10:55all that increased pressure

10:57when they look at lights they will see

10:59halos around the lights

11:00their eyes can be red the pressure can

11:03just be so high that it can cause

11:05edema on the cornea as you can see here

11:07in this picture

11:08and of course they're going to have very

11:10high iop

11:12now let's talk about the key concepts

11:13that you want to know about glaucoma as

11:16the nurse so to help us remember those

11:18key concepts we're going to remember the

11:19word

11:20thief so t is going to be about

11:22treatment goals

11:24and the treatment goal for the glaucoma

11:26of course is to reduce the

11:28intraocular pressure and this is going

11:30to help prevent

11:31damage to the optic nerve so medications

11:33are going to be the first line

11:35treatment for glaucoma most forms of

11:37medications are going to be in eye drops

11:39and

11:40patients can also receive oral meds and

11:42then surgical treatment can be provided

11:45and we're talking about like

11:46laser procedures or traditional eye

11:48surgery

11:49so first laser procedures a type of

11:51laser procedure that can be performed is

11:53called a selective laser

11:56trabeculoplasty also known as an slt

11:59and this procedure is relatively quick

12:01compared to the traditional eye

12:02surgery that we'll talk about next and

12:04it lowers the iop

12:06by using lasers to target certain parts

12:09of that drainage angle

12:10tissue which will cause changes to the

12:13tissue and these changes

12:14will allow that extra aqueous fluid to

12:17drain out of the eye and will decrease

12:20iop now a thing about this is that the

12:23iop

12:24will be lowered over several months and

12:26it's not a permanent

12:28solution next is a more traditional type

12:31of eye surgery and it's typically

12:33performed

12:34when they can't decrease intraocular

12:37pressure in the eyes

12:39because eye drops or other procedures

12:41are not working so a common type of

12:43traditional eye surgery is called a

12:44trabeculectomy

12:46and this is where some of the trabecular

12:48mesh work is removed and an opening is

12:50created to allow aqueous humor to

12:52collect

12:53in an area of the conjunctiva and

12:56this fluid will be reabsorbed and it

12:58will decrease

12:59iop now the patient whenever they have

13:02this procedure will have what's called a

13:04bleb

13:05and you can see that in this picture

13:07here we're pointing to the bleb in

13:09yellow and that is usually going to be

13:11found under

13:12the upper eyelid and this is where that

13:14fluid has collected now important thing

13:17to remember

13:17about these procedures is that vision

13:19that is lost already will not come back

13:22these procedures are going to help

13:23control pressure for several years

13:25and they are not permanent now let's

13:27talk about the post-op education for a

13:29patient who's had glaucoma surgery and

13:32things you need to know as the nurse

13:33so it's very important that you tell the

13:35patient that it's important that they go

13:38to

13:38all of their post-op appointments

13:40because here the doctor is going to be

13:42measuring their eye pressure and looking

13:44at other important post-op

13:45assessments also they don't need to be

13:47driving until they've been cleared by

13:49their doctor

13:50and if they have to go out in direct

13:52sunlight they need to wear protective

13:54sunglasses

13:56also they need to know how to instill

13:58eye drops which we're going to talk

13:59about a little bit later

14:00and to do it exactly as prescribed don't

14:03skip drops

14:05um do some here do some another day it

14:08needs to be exactly how the physician

14:09has ordered it because they're going to

14:10be taking antibiotics

14:11anti-inflammatories etc

14:13and to always use clean hands whenever

14:15they do this plus you need to tell the

14:17patient to refrain from rubbing their

14:18eyes

14:19because they may feel itchy after the

14:21procedure if they have stitches

14:24and they don't want to use their contact

14:26lenses and they want to avoid

14:28activities that increase eye pressure so

14:30you want to make sure that your patient

14:32isn't bending or doing straining

14:34activities like reading

14:36or even straining during a bowel

14:38movement because sometimes patients get

14:40constipated

14:41and they may need stool softeners during

14:43this time because we do not want them

14:45straining to get stool out because this

14:46increases their iop

14:48or lifting heavy objects furthermore you

14:51want to tell them that they need to wear

14:52their eye

14:53shield as prescribed and they may have

14:57blurred vision

14:58their eyes may tear up more frequently

15:00and their eyes may feel itchy

15:02this is normal after the procedure and

15:04this will decrease over time

15:05also they need to report any sudden

15:07vision loss or severe pain

15:09and some pain is common because they've

15:11just had surgery on their eye

15:13and it's very vital they monitor for

15:15infection like any

15:16abnormal discharge a fever extreme eye

15:20redness they can have some

15:22but any extreme or any extreme vision

15:25changes but again blurred vision

15:27at first is normal then we have h for

15:29helpful to remember the three s's

15:31of open angle glaucoma so you definitely

15:34want to know the differences

15:35between open angle and angle closure so

15:38the three s's for open

15:40angle is silent sight stopper so

15:43silent represents the patient's going to

15:45be asymptomatic

15:46until it's too late because the

15:49irreversible damage has been done to

15:51that optic nerve

15:52when the patient actually notices the

15:54loss of peripheral vision

15:55and they have that tunnel vision next is

15:58size so the vision loss can't be brought

16:00back and once blindness occurs it's

16:02permanent and

16:03why does it occur because we've had

16:05increased intraocular pressure that has

16:07damaged

16:08that optic nerve and then the last s

16:10stopper so the development of permanent

16:12vision loss

16:13blindness is stoppable hence it's

16:15avoidable if glaucoma is identified

16:17early through an eye exam that checks

16:19for eye pressure

16:21so let the patient know about this

16:23especially the patients who are at risk

16:25for glaucoma which we're going to talk

16:26about a little bit later

16:28and we can prevent the loss of vision

16:30through medications or procedures that

16:32help keep the iop low

16:34next is eye for immediate treatment for

16:36angle closure glaucoma

16:38so remember this is an emergency and

16:40this is not

16:41a type of glaucoma that's going to be

16:43silent with its signs and symptoms

16:45and medications can be used to lower

16:47that iop like eye drops or oral

16:49medications

16:50along with a surgical procedure like a

16:53laser iridotomy and this is where a

16:56small hole is created in the iris with a

16:59laser to drain that extra aqueous humor

17:02also a more traditional surgery could be

17:05an aerodyctomy and this is where they

17:07remove

17:08parts of the iris and remember the iris

17:10is that colored part of the eyes

17:12so look at those eyeballs it's the green

17:14part and this may be performed in both

17:16eyes because there is a risk of both of

17:18those eyes

17:19actually developing angle closure

17:22glaucoma so they can do that as a

17:23preventative measure

17:24next is e for eye drop so now we're

17:27going to talk about how to administer

17:28eye drops to a patient and i have a

17:30whole video a link should pop up in the

17:32corner

17:33where i actually demonstrate how to do

17:35this but i'm going to go over the

17:36highlights that you need to know

17:38and then we're going to go into the

17:39different types of medications

17:41so first you want to tell the patient

17:43that they don't want to skip days or

17:45abruptly stop

17:46taking the eye drops so some of these

17:49patients they have

17:50quite a few eye drops to take and they

17:52have to take them throughout the day

17:53so it can be hard getting into that

17:55routine and

17:57you want to stress to the patient that

17:59it's very very important that they

18:01take these eye drops exactly as

18:02prescribed because if they skip days

18:04and don't take them here or there they

18:07are ultimately

18:08risking losing their vision because

18:10these drops are made to help

18:11lower their iop also you want to stress

18:14the importance of always washing your

18:16hands before instilling eye drops

18:18because that is one of the big ways we

18:20get sick whenever we

18:22use our hands you know we're touching

18:24surfaces that have

18:25bacteria viruses in them if we touch our

18:28eyes

18:28with those hands we just transfer that

18:31virus or

18:32bacteria into our eye so always use

18:35clean hands when doing this

18:36next is about administering more than

18:38one type of eye drop medication

18:41so as a nurse this is important you know

18:42this and for the patient who's going to

18:44be instilling these at home

18:46so the rule of thumb is to space out

18:48each type of eye drop that the patient

18:50has to take

18:51about three to five minutes so let's say

18:53your patient has two eye drops they need

18:55to take

18:56so they'll take the one eye drop

18:58medication as prescribed and then they

19:00need to wait at least three to five

19:02minutes before instilling the other type

19:04of medication and why do we do this well

19:08we want that first medication to have

19:10time to work to

19:12be in the eye and if we quickly gave

19:14that second

19:15type of eye medication it would just

19:17wash out the first

19:19so you definitely want to have that time

19:22frame between

19:23the types of eye drops and then let's

19:25say your patient has an eye ointment

19:27ordered and an eye drop which one are

19:29you going to give first

19:30you will give the eye drops first and

19:32then you will give the eye ointment

19:34so whenever you give your patient those

19:36eye drops how do you

19:37instill them in the eye well you're

19:39going to place each drop

19:41on the lower sack of the eye and you can

19:43see this sack

19:44here in this picture on the right this

19:46is called the conjunctival sac

19:48you will not directly put the eye drop

19:51on the cornea

19:52via the eye now after instilling each

19:55eye drop medication you want to have the

19:56patient refrain from blinking

19:59but to keep the eye closed and to

20:01perform punctal occlusion

20:03so to do this you will have the patient

20:06or you

20:06use your index finger and gently place

20:09pressure at the side of the bridge of

20:11the nose over

20:13the lacrimal punctum and you can see

20:15that at the top pitcher that's an up

20:17close picture of it

20:18and you'll do this for about two to

20:20three minutes now why

20:22are we doing this well this is to

20:24prevent the medication you just gave

20:26them in their eye

20:27from draining down into the nasolacrimal

20:30duct and being absorbed by the blood

20:33which can cause

20:34widespread signs and symptoms so if you

20:36gave this patient

20:38a commonly prescribed glaucoma

20:40medication like a beta blocker

20:41or a cholinergic eye drop if we didn't

20:45perform this punctal occlusion

20:47that medication that's supposed to stay

20:49in the eye to do its job

20:50can drain down through this tear duct

20:54and can enter into the blood and

20:55actually cause those signs and symptoms

20:57of those medications and we don't want

20:59that to happen

21:00so now let's talk about those

21:01medications so how do medications work

21:04well they're going to help

21:05lower our iop and they're going to do it

21:08one of two ways or both ways they're

21:10going to decrease production of the

21:12aqueous humor

21:14and or they're going to increase the

21:16drainage of the aqueous humor so this is

21:18great we're going to get lower

21:19intraocular pressure so to help us

21:22remember some of these drugs because

21:24there's

21:24quite a few we're going to remember the

21:27abcs so that's going to help us remember

21:29four of them

21:30so first a for alpha agonist and these

21:33tend to end

21:34in i-d-i-n-e like brymotidine

21:37and these work with alpha receptors by

21:40activating them so we'll get the

21:42decrease of aqueous humor production

21:45because it constricts the ciliary body

21:47remember that producer aqueous humor

21:50and this helps increase the drainage of

21:52aqueous humor out of the eye

21:54and lowers our iop now remember this can

21:57cause

21:58systemic effects so we need to do

22:00punctal occlusion

22:01and one side effect that some patients

22:03can have is that they can feel very

22:05drowsy after taking this next is b for

22:08beta blockers and they

22:09end in o l o l like timolol

22:12and these decrease aqueous humor

22:14production and they can cause those

22:16systemic effects majorly so punctual

22:18occlusion very vital

22:20there's different types like

22:21cardio-selective and

22:23non-cardio-selective

22:24and they are not for patients with

22:26bradycardia

22:27asthma or copd and we talked in depth

22:30about this

22:31whenever we talked about our beta

22:32blocker video which you can access

22:34up here in the right corner if you want

22:36to watch that so with this you want to

22:38assess a patient's breathing and heart

22:40rate before administration

22:42then we have c for carbonic anhydrase

22:45inhibitors

22:46and they end in z-o-l-a-m-i-d-e

22:49so zolamides and they can be ordered

22:52orally or via drops

22:54and one medication type is called

22:56acetylzolamide

22:58and these decrease aqueous humor

23:00production so they're like diuretics

23:01that inhibit carbonic acid production

23:04now you want to assess the patient's

23:06allergies because if they have a

23:08sulfonamide allergy they cannot take

23:10these medications

23:12and then our last c is for cholinergics

23:15and these are meiotics and one type is

23:17pilocarpine and how this works is it

23:20helps lower the iop

23:21by constricting the pupil because that's

23:23what these drugs do

23:25and this will help increase the drainage

23:28of aqueous humor

23:29out through that trabecular meshwork now

23:31it's important to be familiar

23:33with the antidote for these drugs which

23:35is atropine

23:36and these drugs can cause that

23:39cholinergic effects like

23:40sweating increased elevation

23:42bronchospasm decreased heart rate

23:44and increase the eye sensitivity with

23:47light

23:48so they may have problems with vision

23:50and dim light

23:51and again it's very important that they

23:53perform punctual

23:54occlusion whenever they take this

23:56medication some other drugs include

23:58a prostaglandin analogues and they end

24:01in pros

24:02like bomatopose and these increase the

24:05drainage of aqueous humor out the eyes

24:07and they don't tend to cause as many

24:09systemic effects as those other

24:10medications that we just went over

24:12but an interesting side effect of this

24:15medication and depends on

24:17if you like this or not it can actually

24:19cause

24:20thicker and longer eyelashes and i have

24:22actually

24:23seen this in a patient it was an older

24:26patient

24:27and their eyelashes were the most

24:28beautiful eyelashes i've ever

24:30seen and i didn't think they were real

24:33and

24:34i looked in their history and they in

24:36fact were taking this medication for the

24:38treatment of glaucoma

24:40and another change that this medication

24:42can cause is that they can change the

24:44color of the iris

24:46to a brown color and then lastly we have

24:48rokinase inhibitors and these

24:50medications are relatively

24:52new for the treatment of glaucoma and

24:55a medication they have is called

24:57natarsadil

24:58and this increases the drainage of

25:00aqueous humor by inhibiting the rock

25:03pathway and this pathway is actually

25:05found in the trabecular mesh work

25:07so it will cause the mesh work to drain

25:09better because remember we talked about

25:11that this mesh work

25:12becomes stiff and it doesn't work very

25:14well

25:15so it'll start to drain and this will

25:17help decrease iop

25:19now one thing that this medication can

25:21cause because it causes dilation

25:23is that it can cause the eyes to become

25:25extremely

25:26red and then the last part of our

25:28mnemonic is f for factors that

25:30increase the development of glaucoma so

25:32to help us remember those risk factors

25:34remember the word save and we're talking

25:36about

25:36save your eyesight so s is for 60 or

25:39older especially if they have a family

25:41history

25:42a is for african american hispanic or

25:44asian community these people

25:46are at risk for glaucoma v for vascular

25:49problems such as like with diabetes

25:51hypertension

25:52and then e for elevated intraocular

25:55pressure because remember

25:56generally that normal iop was 10 to 21

25:59millimeters of mercury

26:01so if they have these risk factors it's

26:03very important that they go for those

26:04eye exams

26:05so they can try to detect if the iop is

26:08elevated

26:08okay so that wraps up this review over

26:11glaucoma and don't forget to access that

26:13free quiz that will test you

26:14on this material

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